Human acclimatization to altitude.
نویسندگان
چکیده
منابع مشابه
Altitude preexposure recommendations for inducing acclimatization.
For many low-altitude (<1500 m) residents, their travel itineraries may cause them to ascend rapidly to high (>2400 m) altitudes without having the time to develop an adequate degree of altitude acclimatization. Prior to departing on these trips, low-altitude residents can induce some degree of altitude acclimatization by ascending to moderate (>1500 m) or high altitudes during either continuou...
متن کاملThromboelastometry and Platelet Function during Acclimatization to High Altitude.
Interaction between hypoxia and coagulation is important given the increased risk of thrombotic diseases in chronically hypoxic patients who reside at sea level and in residents at high altitude. Hypoxia alters the proteome of platelets favouring a prothrombotic phenotype, but studies of activation and consumption of specific coagulation factors in hypoxic humans have yielded conflicting result...
متن کاملRespiratory and circulatory changes during acclimatization to high altitude.
The studies of decreased barometric pressure made by Paul Bert (1) during the latter half of the nineteenth century may properly be considered as the beginning of modern knowledge of, and interest in, the effects of oxygen lack upon the human body. During the same period the rapid development of mountaineering as a sport produced a new occupational disease-altitude sickness, Or seroche as it wa...
متن کاملAcid-base changes associated with respiratory acclimatization to altitude.
The respiratory changes associated with chronic hypoxia are described. The possible biochemical mechanisms which are responsible for the changes in control of ventilation are discussed and relevant experimental evidence assessed.
متن کاملPeripheral chemoreflex function in hyperoxia following ventilatory acclimatization to altitude.
After a period of ventilatory acclimatization to high altitude (VAH), a degree of hyperventilation persists after relief of the hypoxic stimulus. This is likely, in part, to reflect the altered acid-base status, but it may also arise, in part, from the development during VAH of a component of carotid body (CB) activity that cannot be entirely suppressed by hyperoxia. To test this hypothesis, ei...
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ژورنال
عنوان ژورنال: British Journal of Sports Medicine
سال: 1974
ISSN: 0306-3674
DOI: 10.1136/bjsm.8.1.5